My Perspective on ApoB, LDL and Lpa PT 2

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#ldl #lpa #heartattack #heartattacksymptoms #heartdisease #heartdiseasetreatment

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Take a look at the meta analysis from southern Denmark where they calculated longevity based on 11 major statin studies including the Jupiter study and other major studies. This along with various others will convince anyone that statin use does absolutely nothing or may hurt when it considering all cause mortality. Keep in mind and make the adjustment knowing that these studies were done by the pharmaceutical companies and in the case of Jupiter, the co- inventor of the c reactive protein test. Also, many of these studies are done before 2006 where they didn’t have to register the study. It is one thing to read the results of a study using relative risk, but it’s another to dig into it.

jon
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I have had 4 CAC Score scans. On and off statins. Off statins I had a 25% annual increase in my score. On statins I had a 45% annual increase in my score. Statins stabilize plaque by accelerating the calcification of your arteries.

KevinSmith-Liberty
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My lp(a) is 54, fibrogene around 200, cholesterol under 150, HDL 45, Ldl under 90. 2 stents, a lot of sports, 250watt on bike no problem. And still my arteriosclerosis (only in the heart) does not seem to improve. So far no insulin resistence has been detected. As you can see, it's not all cut and dried.

rentner
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It appears that we have much to learn about vascular disease ... 🤔💭
( I like the idea of low dose Crestor as prophylactic to stabilize soft plaque ) ✔️

jamescalifornia
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Could you let me know what do you think about I-P6

glauberborges
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Do you really care what your Lpa is if you are not insulin resistant, fasting insulin less than 4, and metabolically healthy?

jeffrn
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My Aunt was metabolically unhealthy lived to 99 no heart disease but had dementia starting at 88

richardmoeller
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I keep hearing that an increasing calcium score from a statin is good but an increasing calcium score if you aren’t on a statin is bad. Please tell me the difference. It feels like twisted big pharma logic. But maybe I’m wrong.

stevemc
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My body fat is 9.8%, HDL 90 mg/dL, triglyceride 71 mg/dL, LDL 559 mg/dL, total cholesterol 663 mg/dL, remnant cholesterol 14, triglyceride:HDL 0.79, CT Calcium score zero, C Reactive Protein (cardio) 0.7 mg/L, NMR LP+Lipids Small LDL-P <90. BTW, I am a 61 yo LMHR.

MyFatAdaptedLife
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Hey doc what about the 90 year olds that have 350 and up LDL and have no heart disease my doctor told me about one of his patients who is this way.

richardmoeller
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You state in this video that 30-40 is not a significant risk and that even 3-4 times that is not a significant risk. Is that mg/dl or nmol/l?

stacisterling
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My total cholesterol before low carb was 177. Now it's 360. The doctors in Australia think I have FH but my brothers have normal cholesterol and so does my father but my mother has elevated cholesterol but I think because of medicine she takes for depression 🤔

UnknownUser-scjx
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Just got my results for my CAC score, 210. What do I do now?

jeremyking
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How could a practicing doctor NOT know about LP(a) and that it is a big risk factor for cardiac health??? That is scary

jaqueitch
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My LDL is over 300, but I believe I am a LMHR. My most recent blood test showed a Lp-PLA2 of 196 nmol/min/mL supposedly showing reduced risk (under 224). Didn't get my Apo B done this time, but my MPO was 294 pmol/L with low risk under470, and my oxidized LDL was 73 ng/mL (normal range) and (hs)C-reactive protein of 0.19 mg/L, low risk.

lloydhlavac
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Thanks, Dr. But this video goes against the Mendelian randomization studies on LDL-C. People with genetically high LDL-C seem to be at a higher risk independently of insulin resistance

lotembenatar
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If my triglyceride/HDL are 1:1 but my lp(a) is 141, should I be concerned? A1c is 5.4. CRP is 3.44. BMI is around 21.

timsmith