Orbital Apex syndrome

[eclecticism]

I usually fail to distinguish between cavernous sinus syndrome and orbital apex syndrome, one differentiator is that the former despite affecting cranial nerves III, IV, V1, V2, VI and periarterial sympathetic plexus, is usually more acute compared to the subacute course of the latter

[plantinfo]

Behold[or Good Day which ever is received better]: (have a little down time. why not?:: for Idiopathic Orbital Inflammatory Syndrome)The premotor area (PMA), responsible for motor control and coordination, may influence the neural activation of the eye muscles. Excess acetylcholine could lead to receptor saturation, muscle overstimulation, and dysfunction. This could trigger a cascade where the immune system might eventually attack the receptors due to the overstimulation or perceived dysfunction, potentially causing autoimmune-like conditions.

In summary, acetylcholine overproduction impacts the receptor density on the orbital muscles, which is regulated through the PMA as part of the motor control mechanism. - the main mechanism/pathway/~cascade is over production in the body of acetylcholine from excess carbohydrates in the diet which directly increase both receptor cites on the orbital muscles, tissues, and fats, and acetylcholine activation from the receptor neurotransmitter complex, causing inflammation from an over stimulation. lower carbs no unsaturated fat and more vegetables recommended. saturated fat may be increased with stomach closing condiments to slow blood absorption rate/s for nutrients, promoting anabolics and reducing autoimmune related insulin kickback from excessive carbs in the diet. *and no food after 8* that's that for this story..[will work for just about all autoimmune non-genetic dysfucntions, that are related to Acetylcholine production - less or more.]

[sayeedsakib]

will there be nystagmus in orbital apex syndrome?
If not, why?