Understanding LDL Damage: Dr. Paul Mason's Expert Breakdown

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In this video, Dr. Paul Mason talks about LDL Damage

🎙️Dr. Paul Mason is trained Sports and Exercise Medicine Physician with degrees in Medicine, Physiotherapy and Occupational Health. After treating patients with, and lecturing on, low carbohydrate diets for several years, he is now an internationally recognised authority in this field. He firmly believe in the scientific process and will happily challenge medical orthodoxy when warranted by evidence. his clinical practice involves a holistic approach which is informed by evidence rather than conventional thinking. He have special interests in metabolic disease, autoimmune diseases, inflammatory tendon pain and neck & low back pain.

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video credits : @DrPaulMason
video published on 2021

Disclaimer :

This video was created with the aim to educate its audience. Video owner has given us Permission to Use Content. The content on this channel is not medical advice. Please consult your healthcare provider.
Комментарии
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Holy 💩. Higher LDL associated with reversal of plaques.!!! That’s jaw dropping information 👍

rollingstone
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Ketovore improved my lipid panel. After 6 months, my HDL went from 46 mg/dL to 79 mg/dL, my LDL went from 105 mg/dL to 94 mg/dL, and my triglycerides went from 67 mg/dL to 63 mg/dL. I am lean also (20.4 BMI). I ate and continue to eat about 6 eggs for breakfast with about 8 ounces fatty meat, then usually either steak or ground beef for dinner and I maybe have a salad with cheese, bacon and ranch dressing every now and then. I never snack and do about 19/5 or 18/6 IF and occasionally do 36 or 72 hour water only fasts.

RC-tmjo
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It is a pity my cardiologist, and my GP, do not seem to understand this...

gaylerule
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A good point about the vasa vasorum being the likely source of LDL in the artery wall.

arkie
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He's referring to the NMR LDL-P that measures particle size and concentration. Pheno type A is associated with larger LDL particles. LDL-P correlates with HDL TRIGLYCERIDE RATIO.

MK-ftqt
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This is good video for people that doesn´t know anything of cholesterol. Doctor wanted to put me on statins becouse my LDL is higher than most of the people or as them book says. I said to him that i don´t have insuline resistance, t2d or inflammation after being 15 months on keto. So i don´t have the risk factors for oxodized LDL. He mumbled for while and sounded angry that i resisted of taking unnecessary pills to treat my cholesterol even that there isn´t any problem. At least here in Finland they don´t take larger scale tests that what kind of LDL you have.
Also the heart hospitals guide lines from nutrition is horrible. Same goes with Finnish diabetes association. Just like ADA in US gives such a horrible knowledge of the nutrition, i don´t wonder why people doesn´t ever get cured from those diseases.

ArchieArpeggio
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THANK You...! 😊👍 Very Enlightening and Comprehensive -70SomethingGuy

jarichardsutube
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The higher the insulin resistance the higher the blood glucose which leads to higher glycation of LDL. This will result in higher than normal sdLDL. As a layperson, this is how I understood it. Please correct me if I am wrong.

heping
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One who knows his subject explains it well.

goodviewfromuphere
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Very clear explanation thanks. My LDL is fine. Will live longer. 😉

AnneMB
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I'd love to see a citaation that supports the claim that high LDL individuals seem to have a better chance of plaque regression.

chazwyman
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Can you point to the studies that are mentioned? :) Especially the plaque reversals. Thanks!

ap-performance
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Regarding the higher ldl patients had plaque regression, could you please provide the reference?

kyefang
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4:50 anyone knows this observational study and has thr link?

ChefJollyRoger
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1:00 I am Jeff. I am not a bus. I am a carnivore. 🙂

JeffSanfordJr
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Question is: why can’t VLDL and/or IDL not oxydise? They don’t have an APOB-100 molecule? Does that APOB-100 suddenly appear on a LDL particle. And what causes oxidation?

magicf
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Could the thyroid function be at root of high cholesterol.?

lizakiesling
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My GP (over the phone ) suggested statins as my total Chol was 6 mmmol/l. She seemed baffled when I said "Based on one biomarker in an otherwise healthy person you would prescribe a life-changing drug?"
I then asked how long I would be on this drug? Answer: Until you're 80!
Huh, as age is a risk factor for CVD, then I would be more at risk at 80 years of age!
I ended this ridiculous conversation with "I'll do my research".

EricaNernie
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Why would you have oxidized LDL if you do NOT eat sugar or seed oils? Are there other risk factors one can work on? Can the fats in whole nuts and seeds also be damaged?

ML-yfdl
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ApoB100 - the address for re-uptake of LDL by the liver. Oxidized LDL cannot be absorbed by the liver. I believe inflammation is also evidently a huge culprit when it gets to oxidation and damage to the LDL.
My last blood works look pretty good, but as in 2023, my CRP levels remain stubbornly high; around 10 mg/l.
My trigs are fine (I was not fasted) and my HDL-C is a super high 1.9 mmol/l.

Straightdeal